目前作为课题负责人主持国家青年基金1项，国家十二五重大专项子课题1项，四川省卫生厅课题1项，作为第一主研参与国家自然基金面上项目一项，同时还参与国家科技部“十一五”“十二五”艾滋病及病毒性肝炎重大传染病防治重大专项，“973”计划2项。发表科研论文及综述30余篇，目前在Molecular Cancer Therapeutics，Journal of Cell Biochemistry， Molecular Pharmacology，Apoptosis等期刊收录的英文杂志上发表论文10余篇，其中第一作者5篇（影响因子在5.0以上的杂志1篇）。参与完成的课题获中华医学科技奖三等奖一项，四川省科技进步奖二等奖一项。近期发表文章如下：1.Bai L, Xu X, Wang X, Xu S, Ju W, Chen W, He W, Wang Q, Tang H, Lin Y. A superoxide-mediated MKP-1 degeneration and JNK activation pathway for luteolin-induced lung cancer cytotoxicity.. Mol Pharmacol. 2012;81:549—555(影响因子：4.8)2．X Xu.L., Bai L., Chen W.S., Mabel T. Padilla, Liu YS, Kim KC, Belinsky SA, Lin Y.. Muc-1 contributes to BPDE-induced human bronchial epithelial cell trandformation through facilitating EGFR activation.Plos one.7(3):e33846,doi:10.1371/journal.pone. 0033846(影响因子：4.08)3. Bai L, Xu S, Chen W, Li Z, Wang X, Tang H, Lin Y. Blocking NF-κB and Akt by Hsp90 inhibition sensitizes Smac mimetic compound 3-induced extrinsic apoptosis pathway and results in synergistic cancer cell death. Apotosis. 2011;16(1):45-54 (影响因子：4.066)4. Bai L, Chen WS, Chen WJ, Wang X, Tang H, Lin Y. IKKβ-mediated nuclear factor- B activation attenuates smac mimetic–induced apoptosis in cancer cells. Mol. Cancer Ther. 2009; 8(6):1636-1645 (影响因子：5.003)5. Bai L, Chen W, Wang X, Ju W, Xu S, Lin Y. Attenuating Smac mimetic compound 3-induced NF-kappaB activation by luteolin leads to synergistic cytotoxicity in cancer cells. J Cell Biochem. 2009;108(5):1125-1131(影响因子：3.54) 6.Chen WJ, Bai L, Wang X, Xu S, Belinsky SA, Lin Y. Acquired activation of the Akt/Cyclooxygenase-2/Mcl-1 Pathway Renders Lung Cancer Cells Resistant to Apoptosis Mol. Pharmacol. 2010;77(3):416-423(影响因子：4.711)7. Lin Y, Bai L, Chen W, Xu S.The NF-kappaB activation pathways, emerging molecular targets for cancer prevention and therapy. Expert Opin Ther Targets.2010;14(1):45-55 (影响因子：4.038)8. Chen W, Li Z, Bai L, Lin Y. NF-kappaB in lung cancer, a carcinogenesis mediator and a prevention and
